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The contraction of the heart ventricles is driven by myocyte shortening. Being microstructurally anchored, directly linked to mechanical activation, and independent of arbitrary (cylindrical) reference systems, aggregate cardiomyocytes strain appears as an ideal candidate to measure cardiac function in healthy and diseased hearts. Our goal is to compute aggregate cardiomyocyte shortening based on computational models and cardiac MRI images.


Luigi Perotti, Ph.D.
Assistant Professor of Mechanical and Aerospace Engineering